The library will be closed between August 10, 2026 and August 16, 2026. During this period, it will be possible to upload publications, but all other services will be suspended.
Tzerpos, P.,
Bojcsuk, D.,
Bene, K.,
Caballero-Sánchez, N.,
Parteka-Tojek, Z.,
Banecki, K.,
Korsak, S.,
Pritchett, S. M.,
Patsalos, A.,
Oleksak, D.,
Berger, W. K.,
Sarris, M. E.,
Giannakis, N.,
Chantalat, S.,
Silye-Cseh, T.,
Papachristou, E. K.,
Erdélyi, F.,
Máté, Z.,
Szabó, G.,
Nagy, G.,
Carroll, J. S.,
Spilianakis, C. G.,
Schwabe, J. W. R.,
Plewczynski, D.,
Nagy, L.:
BACH1 acts as a pioneer repressor to enable macrophage plasticity and adaptation across tissues and inflammatory contexts.
Immunity. "Accepted by Publisher", 2026.
Buete, R.,
Scherer, J.,
Patsalos, A.,
Nagy, L.:
From lipid switch to tissue repair: how resolvins reprogram macrophage polarization and function.
Immunometabolism 8 (1), 1-9, (article identifier: 00076), 2026.
Kolostyák, Z.,
Nagy, L.:
The gene regulatory networks shaping macrophage plasticity and altered function in fibrosis.
Front. Immunol. 17, 1-26, (article identifier: 1767954), 2026.
Kiss, M.,
Halász, L.,
Hadadi, É.,
Berger, W. K.,
Tzerpos, P.,
Póliska, S.,
Kancheva, D.,
Gabriel, A.,
Barthelmess, R. M.,
Debraekeleer, A.,
Brughmans, J.,
Elkrim, Y.,
Martens, L.,
Saeys, Y.,
Dániel, B.,
Czimmerer, Z.,
Laoui, D.,
Nagy, L.,
Van Ginderachter, J. A.:
Epigenomic preconditioning of peripheral monocytes determines their transcriptional response to the tumor microenvironment.
Genome Med. 17 (1), 1-17, (article identifier: 82), 2025.
Halász, H. E.,
Papp, A. B.,
Lesinszki, L. S.,
Mócsai, A.,
Varga, T.,
Nagy, L.,
Gogolák, P.:
Neutrophil deficiency increases T-cell numbers at the site of tissue injury in mice.
FEBS Lett. 599 (18), 2644-2662, 2025.
Zhang, L.,
Han, C.,
Shrestha, M. M.,
Le, J.,
Berger, W. K.,
Huang, Y.,
Desrouleaux, R.,
Wang, E.,
Nagy, L.,
Yang, X.:
The OGT-TFF2 axis mediates intrahepatic crosstalk and MASH pathogenesis.
Hepatology. [Epub ahead of print], 2025.
Baráth, B. R.,
Nagy, L.:
A serine metabolic enzyme is flexing its muscle to help repair skeletal muscle.
Genes Dev. 38 (3-4), 95-97, 2024.
(Reviewed publication: Veronica Ciuffoli, Xuesong Feng, Kan Jiang, Natalia Acevedo-Luna, Kyung Dae Ko, A. Hong Jun Wang, Giulia Riparini, Mamduh Khateb, Brian Glancy, Stefania Dell'Orso, Vittorio Sartorelli. -Psat1-generated a-ketoglutarate and glutamine promote muscle stem cell activation and regeneration /In: Genes & Development. - Megjelenés alatt (2024). -)
Tordai, C.,
Hathy, E.,
Gyergyák, H.,
Vincze, K.,
Baradits, M.,
Koller, J.,
Póti, Á.,
Jezsó, B.,
Homolya, L.,
Molnár, M. J.,
Nagy, L.,
Szüts, D.,
Apáti, Á.,
Réthelyi, J.:
Probing the biological consequences of a previously undescribed de novo mutation of ZMYND11 in a schizophrenia patient by CRISPR genome editing and induced pluripotent stem cell based in vitro disease-modeling.
Schizophr. Res. 273, 107-120, 2024.
Caballero-Sánchez, N.,
Alonso-Alonso, S.,
Nagy, L.:
Regenerative inflammation: When immune cells help to re-build tissues.
FEBS J. 291 (8), 1593-1614, 2024.
Patsalos, A.,
Halász, L.,
Oleksak, D.,
Wei, X.,
Nagy, G.,
Tzerpos, P.,
Conrad, T.,
Hammers, D. W.,
Sweeney, H. L.,
Nagy, L.:
Spatiotemporal transcriptomic mapping of regenerative inflammation in skeletal muscle reveals a dynamic multilayered tissue architecture.
J. Clin. Invest. 134 (20), 1-20, (article identifier: 173858), 2024.
Caballero-Sánchez, N.,
Winn, N.,
Rosa Neto, J. C.,
Nagy, L.:
Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism.
Front Endocrinol (Lausanne). 14, 1-2, (article identifier: 1219036), 2023.
Caballero-Sánchez, N.,
Winn, N.,
Neto, J. C. R.,
Nagy, L.:
Editorial: Cross-talk between heterogeneous cell types in skeletal muscle : implications for glucose metabolism.
Front Endocrinol (Lausanne). 14, 1-3, (article identifier: 1185725), 2023.
Patsalos, A.,
Halász, L.,
Medina-Serpas, M. A.,
Berger, W. K.,
Dániel, B.,
Tzerpos, P.,
Kiss, M.,
Nagy, G.,
Fischer, C.,
Simándi, Z.,
Varga, T.,
Nagy, L.:
A growth factor-expressing macrophage subpopulation orchestrates regenerative inflammation via GDF-15.
J. Exp. Med. 219 (1), 1-38, (article identifier: 20210420), 2022.
