Bódi Beáta

1.

Ráduly, A. P., Nagy, L. T., Bódi, B., Papp, Z., Borbély, A.: Biomarkers in heart failure: Traditional and emerging indicators for prognosis.
ESC Heart Failure. [Epub ahead of print], 2024.

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2.

Szőke, K., Bódi, B., Hendrik, Z., Czompa, A., Gyöngyösi, A., Haines, D. D., Papp, Z., Tósaki, Á., Lekli, I.: Rapamycin treatment increases survival, autophagy biomarkers and expression of the anti-aging klotho protein in elderly mice.
Pharmacol. Res. & Perspec. 11 (3), 1-11, (article identifier: 01091), 2023.

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Q2 Neurology

Q1 Pharmacology, Toxicology and Pharmaceutics (miscellaneous)

3.

Bódi, B., Pilz, P. M., Mártha, L., Lang, M., Hamza, O., Fagyas, M., Szabó, P. L., Abraham, D., Tóth, A., Podesser, B. K., Kiss, A., Papp, Z.: Alterations in ACE and ACE2 Activities and Cardiomyocyte Signaling Underlie Improved Myocardial Function in a Rat Model of Repeated Remote Ischemic Conditioning.
Int. J. Mol. Sci. 22 (20), 1-17, 2021.

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Q2 Catalysis

Q1 Computer Science Applications

Q1 Inorganic Chemistry

Q1 Medicine (miscellaneous)

Q2 Molecular Biology

Q1 Organic Chemistry

Q1 Physical and Theoretical Chemistry

D1 Spectroscopy

4.

Bódi, B., Oláh, A., Mártha, L., Tóth, A., Radovits, T., Merkely, B., Papp, Z.: Exercise-induced alterations of myocardial sarcomere dynamics are associated with hypophosphorylation of cardiac troponin I.
Reviews in Cardiovascular Medicine. 22 (4), 1079-1085, 2021.

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Q2 Cardiology and Cardiovascular Medicine

Q2 Medicine (miscellaneous)

5.

Kovács, Á., Herwig, M., Budde, H., Delalat, S., Kolijn, D., Bódi, B., Hassoun, R., Tangos, M., Zhazykbayeva, S., Balogh, Á., Czuriga, D., Linthout, S. V., Tschöpe, C., Dhalla, N. S., Mügge, A., Tóth, A., Papp, Z., Barta, J., Hamdani, N.: Interventricular Differences of Signaling Pathways-Mediated Regulation of Cardiomyocyte Function in Response to High Oxidative Stress in the Post-Ischemic Failing Rat Heart.
Antioxidants. 10 (6), 1-21, 2021.

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Q2 Biochemistry

Q2 Cell Biology

Q1 Clinical Biochemistry

Q2 Molecular Biology

Q2 Physiology

6.

Bódi, B., Kovács, Á., Gulyás, H., Mártha, L., Tóth, A., Mátyás, C., Barta, B. A., Oláh, A., Merkely, B., Radovits, T., Papp, Z.: Long-Term PDE-5A Inhibition Improves Myofilament Function in Left and Right Ventricular Cardiomyocytes through Partially Different Mechanisms in Diabetic Rat Hearts.
Antioxidants. 10 (11), 1-13, 2021.

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Q2 Biochemistry

Q2 Cell Biology

Q1 Clinical Biochemistry

Q2 Molecular Biology

Q2 Physiology

7.

Fülöp, G. Á., Oláh, A., Csípő, T., Kovács, Á., Pórszász, R., Veress, R., Horváth, B., Nagy, L., Bódi, B., Fagyas, M., Helgadottir, S. L., Bánhegyi, V., Juhász, B., Bombicz, M., Priksz, D., Nánási, P. P. i., Merkely, B., Édes, I., Csanádi, Z., Papp, Z., Radovits, T., Tóth, A.: Omecamtiv mecarbil evokes diastolic dysfunction and leads to periodic electromechanical alternans.
Basic Res. Cardiol. 116 (1), 24, 2021.

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Q1 Cardiology and Cardiovascular Medicine

Q1 Physiology

Q1 Physiology (medical)

8.

Lódi, M., Bánhegyi, V., Bódi, B., Gyöngyösi, A., Kovács, Á., Árokszállási, A., Hamdani, N., Fagyas, M., Édes, I., Csanádi, Z., Czuriga, I., Kisvárday, Z., Lekli, I., Bai, P., Tóth, A., Papp, Z., Czuriga, D.: Prophylactic, single-drug cardioprotection in a comparative, experimental study of doxorubicin-induced cardiomyopathy.
J. Transl. Med. 18 (1), 470, 2020.

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Q1 Biochemistry, Genetics and Molecular Biology (miscellaneous)

D1 Medicine (miscellaneous)

9.

Lódi, M., Priksz, D., Fülöp, G. Á., Bódi, B., Gyöngyösi, A., Nagy, L., Kovács, Á., Kertész, A. B., Kocsis, J., Édes, I., Csanádi, Z., Czuriga, I., Kisvárday, Z., Juhász, B., Lekli, I., Bai, P., Tóth, A., Papp, Z., Czuriga, D.: Advantages of prophylactic versus conventionally scheduled heart failure therapy in an experimental model of doxorubicin-induced cardiomyopathy.
J. Transl. Med. 17, 229, 2019.

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Q1 Biochemistry, Genetics and Molecular Biology (miscellaneous)

D1 Medicine (miscellaneous)

10.

Ruppert, M., Bódi, B., Nagy, D., Korkmaz-Icöz, S., Loganathan, S., Oláh, A., Barta, B. A., Sayour, A. A., Benke, K., Karck, M., Merkely, B., Papp, Z., Szabó, G., Radovits, T.: A miofilamentáris rendszer Ca2+-érzékenysége korrellál a bal kamrai kontraktilitással a fokozott nyomásterhelés által előidézett patológiás szívizom-hipertrófia patkánymodelljében.
Cardiol. Hung. 49 (2), 88-99, 2019.

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11.

Ruppert, M., Bódi, B., Korkmaz-Icöz, S., Loganathan, S., Jiang, W., Lehmann, L., Oláh, A., Barta, B. A., Sayour, A. A., Merkely, B., Karck, M., Papp, Z., Szabó, G., Radovits, T.: Myofilament Ca2+ sensitivity correlates with left ventricular contractility during the progression of pressure overload-induced left ventricular myocardial hypertrophy in rats.
J. Mol. Cell. Cardiol. 129, 208-218, 2019.

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Q1 Cardiology and Cardiovascular Medicine

Q2 Molecular Biology

12.

Márton, J., Péter, M., Balogh, G., Bódi, B., Vida, A., Szántó, M., Bojcsuk, D., Jankó, L., Bhattoa, H. P., Gombos, I., Uray, K., Horváth, I., Török, Z., Bálint, B. L., Papp, Z., Vígh, L., Bai, P.: Poly(ADP-ribose) polymerase-2 is a lipid-modulated modulator of muscular lipid homeostasis.
Biochim. Biophys. Acta. Mol. Cell Biol. Lipids. 1863 (11), 1399-1412, 2018.

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Q1 Cell Biology

Q1 Molecular Biology

13.

Nagy, L., Gődény, I., Nánási, P. P. i., Leskó, Á., Balogh, L., Bánhegyi, V., Bódi, B., Csípő, T., Csongrádi, A., Fülöp, G. Á., Kovács, Á., Lódi, M., Papp, Z.: A szív pozitív inotróp támogatása a miozin-aktivátor hatású omecamtiv-mecarbil segítségével.
Cardiol. Hung. 47 (1), 69-76, 2017.

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14.

Mátyás, C., Németh, B. T., Oláh, A., Török, M., Ruppert, M., Kellermayer, D., Barta, B. A., Szabó, G., Kökény, G., Horváth, E. M., Bódi, B., Papp, Z., Merkely, B., Radovits, T.: Prevention of the development of heart failure with preserved ejection fraction by the phosphodiesterase-5A inhibitor vardenafil in rats with type 2 diabetes.
Eur. J. Heart Fail. 19 (3), 326-336, 2017.

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D1 Cardiology and Cardiovascular Medicine

15.

Bódi, B., Pásztorné Tóth, E., Nagy, L., Tóth, A., Mártha, L., Kovács, Á., Balla, G., Kovács, T., Papp, Z.: Titin isoforms are increasingly protected against oxidative modifications in developing rat cardiomyocytes.
Free Radic. Biol. Med. 113, 224-235, 2017.

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Q1 Biochemistry

Q1 Physiology (medical)

16.

Kovács, Á., Fülöp, G. Á., Kovács, A., Csípő, T., Bódi, B., Priksz, D., Juhász, B., Beke, L., Hendrik, Z., Méhes, G., Granzier, H. L., Édes, I., Fagyas, M., Papp, Z., Barta, J., Tóth, A.: Renin overexpression leads to increased titin-based stiffness contributing to diastolic dysfunction in hypertensive mRen2 rats.
Am. J. Physiol.-Heart Circul. Physiol. 310 (11), H1671-H1682, 2016.

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Q1 Cardiology and Cardiovascular Medicine

Q1 Physiology

Q1 Physiology (medical)

17.

Nagy, L., Kovács, Á., Bódi, B., Pásztorné Tóth, E., Fülöp, G. Á., Tóth, A., Édes, I., Papp, Z.: The novel cardiac myosin activator omecamtiv mecarbil increases the calcium sensitivity of force production in isolated cardiomyocytes and skeletal muscle fibres of the rat.
Br. J. Pharmacol. 172 (18), 4506-4518, 2015.

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D1 Pharmacology

18.

Kalász, J., Pásztorné Tóth, E., Bódi, B., Fagyas, M., Tóth, A., Bhattoa, H. P., Vári, S. G., Balog, M., Blažetić, S., Heffer, M., Papp, Z., Borbély, A.: Single acute stress-induced progesterone and ovariectomy alter cardiomyocyte contractile function in female rats.
Croat. Med. J. 55 (3), 239-249, 2014.

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Q2 Medicine (miscellaneous)

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