Kovács Adrienn

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2014-2023

2023

Csemer, A., Kovács, A., Maamrah, B., Pocsai, K., Korpás, K. L., Klekner, Á., Szűcs, P., Nánási, P. P., Pál, B.: Astrocyte- and NMDA receptor-dependent slow inward currents differently contribute to synaptic plasticity in an age-dependent manner in mouse and human neocortex.
Aging Cell. 22 (9), e13939, (article identifier: 13939), 2023.

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D1 Aging

Q1 Cell Biology

2021

Bayasgalan, T., Stupniki, S., Kovács, A., Csemer, A., Szentesi, P., Pocsai, K., Dionisio, L., Spitzmaul, G., Pál, B.: Alteration of mesopontine cholinergic function by the lack of KCNQ4 subunit.
Front. Cell. Neurosci. 15, 707789, 2021.

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Q1 Cellular and Molecular Neuroscience

Dautan, D., Kovács, A., Bayasgalan, T., Diaz-Acevedo, M. A., Pál, B., Mena-Segovia, J.: Modulation of motor behavior by the mesencephalic locomotor region.
Cell Reports. 36 (8), 109594, 2021.

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D1 Biochemistry, Genetics and Molecular Biology (miscellaneous)

Bayasgalan, T., Csemer, A., Kovács, A., Pocsai, K., Pál, B.: Topographical Organization of M-Current on Dorsal and Median Raphe Serotonergic Neurons.
Front. Cell. Neurosci. 15, 614947, 2021.

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Q1 Cellular and Molecular Neuroscience

2019

Baksa, B., Kovács, A., Bayasgalan, T., Szentesi, P., Kőszeghy, Á., Szűcs, P., Pál, B.: Characterization of functional subgroups among genetically identified cholinergic neurons in the pedunculopontine nucleus.
Cell. Mol. Life Sci. 76 (14), 2799-2815, 2019.

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Q1 Cell Biology

D1 Cellular and Molecular Neuroscience

Q1 Molecular Biology

D1 Molecular Medicine

D1 Pharmacology

Kovács, A., Baksa, B., Bayasgalan, T., Szentesi, P., Csemer, A., Pál, B.: Orexinergic actions modify occurrence of slow inward currents on neurons in the pedunculopontine nucleus.
Neuroreport. 30 (14), 933-938, 2019.

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Q3 Neuroscience (miscellaneous)

2017

Kovács, A., Pál, B.: Astrocyte-Dependent Slow Inward Currents (SICs) Participate in Neuromodulatory Mechanisms in the Pedunculopontine Nucleus (PPN).
Front. Cell. Neurosci. 11 (16), 1-16, 2017.

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Q1 Cellular and Molecular Neuroscience

Kovács, A., Bordás, C., Bíró, T., Hegyi, Z., Antal, M., Szűcs, P., Pál, B.: Direct presynaptic and indirect astrocyte-mediated mechanisms both contribute to endocannabinoid signaling in the pedunculopontine nucleus of mice.
Brain Struct. Funct. 222 (1), 247-266, 2017.

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D1 Anatomy

D1 Histology

Q1 Neuroscience (miscellaneous)

2016

Oláh, T., Bodnár, D., Tóth, A., Vincze, J., Fodor, J., Reischl, B., Kovács, A., Ruzsnavszky, O., Dienes, B., Szentesi, P., Friedrich, O., Csernoch, L.: Cannabinoid signalling inhibits sarcoplasmic Ca2+ release and regulates excitation-contraction coupling in mammalian skeletal muscle.
J. Physiol.-London. 594 (24), 7381-7398, 2016.

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Q1 Physiology

D1 Sports Science

2015

10.

Oláh, T., Bodnár, D., Tóth, A., Fodor, J., Kovács, A., Farkas, A., Nádró, B., Szentesi, P., Csernoch, L.: CB1 cannabinoid receptors are involved in the regulation of excitation-contraction coupling in mammalian skeletal muscle.
J. Muscle Res. Cell. Motil. 36, 129, 2015.

doi DEA

11.

Kovács, A., Bordás, C., Pál, B.: Cholinergic and endocannabinoid neuromodulatory effects overlap on neurons of the pedunculopontine nucleus of mice.
Neuroreport. 26 (5), 273-278, 2015.

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Q3 Neuroscience (miscellaneous)

12.

Kőszeghy, Á., Kovács, A., Bíró, T., Szűcs, P., Vincze, J., Hegyi, Z., Antal, M., Pál, B.: Endocannabinoid signaling modulates neurons of the pedunculopontine nucleus (PPN) via astrocytes..
Brain. Struct. Funct. 220 (5), 3023-3041, 2015.

doi DEA

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D1 Anatomy

D1 Histology

Q1 Neuroscience (miscellaneous)

13.

Bordás, C., Kovács, A., Pál, B.: The M-current contributes to high threshold membrane potential oscillations in a cell type-specific way in the pedunculopontine nucleus of mice.
Front. Cell. Neurosci. 9, 2015.

doi DEA

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Q1 Cellular and Molecular Neuroscience

2014

14.

Oláh, T., Bodnár, D., Tóth, A., Fodor, J., Kovács, A., Farkas, A., Nádró, B., Szentesi, P., Csernoch, L.: Cannabinoids and muscle weakness - Investigating the function of CB1 receptors in mammalian skeletal muscle.
Acta Physiol. 211 (Suppl), 82-83, 2014.

doi DEA