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Q1
Biochemistry, Genetics and Molecular Biology (miscellaneous)
2019
2.
Sághy, T.,
Köröskényi, K.,
Hegedűs, K.,
Antal, M.,
Bankó, C.,
Bacsó, Z.,
Papp, A.,
Stienstra, R.,
Szondy, Z.:
Loss of Transglutaminase 2 Sensitizes for DietInduced Obesity-Related Inflammation and Insulin Resistance due to Enhanced Macrophage c-Src Signaling.
Cell Death Dis. 10, 1-14, (article identifier: 439), 2019.
Yen, J. H.,
Lin, L. C.,
Chen, M. C.,
Sarang, Z.,
Leong, P. Y.,
Chang, I. C.,
Hsu, J. D.,
Chen, J. H.,
Hsieh, Y. F.,
Pallai, A.,
Köröskényi, K.,
Szondy, Z.,
Tsay, G. J.:
The metastatic tumor antigen 1-transglutaminase-2 pathway is involved in self-limitation of monosodium urate crystal-induced inflammation by upregulating TGF-[béta]1.
Arthritis Res Ther. 17 (65), 1-13, 2015.
Duró, E.,
Pallai, A.,
Köröskényi, K.,
Sarang, Z.,
Szondy, Z.:
Adenosine A3 receptors negatively regulate the engulfment-dependent apoptotic cell suppression of inflammation.
Immunol. Lett. 162 (2), 292-301, 2014.
Mihály, J.,
Marosvölgyi, T.,
Szegedi, A.,
Köröskényi, K.,
Lucas, R.,
Töröcsik, D.,
Garcia, A. L.,
Decsi, T.,
Rühl, R.:
Increased FADS2-Derived n-6 PUFAs and Reduced n-3 PUFAs in Plasma of Atopic Dermatitis Patients.
Skin Pharmacol. Physiol. 27 (5), 242-248, 2014.
Keresztesi, E.,
Köröskényi, K.,
Fésüs, L.,
Szondy, Z.:
Adenosine As A Mediator Of The Antiinflammatory Effects Of Apoptotic Cell Uptake.
In: FEBS3 + Meeting From molecules to life and back, Book of abstracts. Eds.: Jerka Dumic, Zrinka Kovarik, Jadranka Varljen, Croatian Society of Biochemistry and Molecular Biology, Opatija, Croatia, 205, 2012. ISBN: 9789539555144
Köröskényi, K.,
Duró, E.,
Pallai, A.,
Sarang, Z.,
Kloor, D.,
Ucker, D. S.,
Beceiro, S.,
Castrillo, A.,
Chawla, A.,
Ledent, C. A.,
Fésüs, L.,
Szondy, Z.:
Involvement of adenosine A2A receptors in engulfment-dependent apoptotic cell suppression of inflammation..
J. Immunol. 186 (12), 7144-7155, 2011.
Sarang, Z.,
Köröskényi, K.,
Pallai, A.,
Duró, E.,
Melino, G.,
Griffin, M.,
Fésüs, L.,
Szondy, Z.:
Transglutaminase 2 null macrophages respond to lipopolysaccharide stimulation by elevated proinflammatory cytokine production due to an enhanced [alfa](v)[béta](3) integrin-induced Src tyrosine kinase signaling.
Immunol. Lett. 138 (1), 71-78, 2011.